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Dihydrocodeine is a semi-synthetic narcotic analgesic with potency between morphine and codeine. In mice the ED50 for analgesic effect after s.c. administration was 12.4mg/kg compared with morphine (2.1mg/kg) and codeine (14.2mg/kg). Duration of analgesia was about 2 hours for morphine and dihydrocodeine and about 1 hour for codeine. Where to and how can I buy Dihydrocodeine Continus 30mg, 60mg, 90mg, 120mg tablets online over the counter without prescription in uk next day delivery.

Besides analgesia, dihydrocodeine has an antitussive effect (depression of the cough reflex by direct effect on the cough centre in the medulla). Antitussive effects may occur with dosages lower than those usually required for analgesia. Dihydrocodeine also causes a reduction in motility of gastrointestinal smooth muscle with an increase in tone in the antrum of the stomach and duodenum. Digestion of food in the small intestine is delayed and propulsive contractions are decreased. Propulsive peristaltic waves in the colon are decreased, while tone is increased to the point of spasm resulting in constipation. Where to and how can I buy Dihydrocodeine Continus 30mg, 60mg, 90mg, 120mg tablets online over the counter without prescription in uk next day delivery.

Dihydrocodeine acts on opioid receptors in the brain to reduce the patient’s perception of pain and improve the psychological reaction to the pain by reducing the associated anxiety. Unlike morphine, dihydrocodeine is not a full agonist. Increasing the human dose beyond 240mg/day does not produce useful additional analgesic effect.

Through direct action on the central nervous system, dihydrocodeine may also produce drowsiness, mental clouding and mood alteration (euphoria or dysphoria). Where to and how can I buy Dihydrocodeine Continus 30mg, 60mg, 90mg, 120mg tablets online over the counter without prescription in uk next day delivery.

Such effects may be common at first but tolerance develops with prolonged administration. Other centrally mediated effects that are normally of low incidence at therapeutic doses include respiratory depression, nausea and vomiting. Respiratory depression results from reduced responsiveness of the respiratory centre to carbon dioxide. Nausea and vomiting are a consequence of direct stimulation of the chemoreceptor trigger zone.